Trọng Hiếu, Hoàng Tôn
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Lời bài hát Souvenir – Trọng Hiếu, Hoàng Tôn

Souvenir - Trọng Hiếu, Hoàng Tôn

Souvenir - Trọng Hiếu, Hoàng Tôn

Thông tin bài hát

Tên bài hát: Souvenir
Ca sĩ: Trọng Hiếu, Hoàng Tôn
Sáng tác: Hoàng Tôn, Trọng Hiếu
Album: Yêu Chill (EP)
Ngày ra mắt: 25/10/2023
Thể loại: Việt Nam, R&B Việt

 

Lời bài hát Souvenir – Trọng Hiếu, Hoàng Tôn

Anh muốn đi khỏi nơi này
Để nỗi buồn qua
Anh muốn nói mà lại để
Cơn gió vụt qua
Trong ánh mắt của em sao mây
You can take my breath away
Giờ xung quanh chỉ toàn mờ mây
Yeah it’s true I don’t know what to do
Vì nghĩ đến em vì nghĩ đến em
Damn right it’s true
I don’t know what to do
Chỉ mong là có em ở đây
Something about you
Make me feel brand new
Bây giờ anh chỉ muốn biết
Baby do you like me too
I can take your soul
You can keep my heart
Và đừng xem nó
Là vật trang sức hay souvenir
Whoo Whoo
Và đừng xem nó
Là vật trang sức hay souvenir
Whoo Whoo
Và đừng xem nó là souvenir
Ngay lần đầu mà ta đã gặp nhau
Anh đã biết là chẳng thể nào
Mà quên được đâu
Đi vào hơi sâu
Nói chuyện hơi lâu
Không phải nam châm
Mà hút nhau như là ngược dấu
Xoay vòng em thì có thật nhiều
Đàn ông anh biết mà
Nhưng mà ai làm em phải rên
Và la em biết mà
Bữa tiệc long lanh
Vui vẻ không phanh
Đó là cách của anh
Để cho em yêu biết là
Anh chẳng quan tâm ngoài kia những thị phi
Đưa em vào store Louis V
Em có thể lấy hết
Tất cả thứ em muốn
Nhưng đừng khiến con tim anh nó phải suy
Vì khi yêu thì ai cũng đâm đầu babe
Nghĩ về em hết đêm ngày babe
Whoo Whoo
Something about you
Make me feel brand new
Bây giờ anh chỉ muốn biết
Baby do you like me too
I can take your soul
You can keep my heart
Và đừng xem nó
Là vật trang sức hay souvenir
There’s something about you
Make me feel brand new
Bây giờ anh chỉ muốn biết
Baby do you like me too
I can keep your soul
You can keep my heart
Đừng làm mất souvenir
Whoo

63 thoughts on “Lời bài hát Souvenir – Trọng Hiếu, Hoàng Tôn”

  56. steroids winstrol says:

    Regardless, complete research using refined methodologies to precisely measure serum MSTN concentrations is crucial to finest
    decide if lowered MSTN impacts the effectiveness of inhibition therapies [103, 104].
    Electrical stimulation of the soleus muscle was performed to measure
    power, fatigue, and recovery from fatigue following all treatments.
    A detailed description of soleus electrophysiology experiments is described in Hanson et al.9.

    Methoxyisoflavone (M), 20-hydroxyecdysone (E), and sulfo-polysaccharide (CSP3) have been marketed to athletes as dietary supplements that
    can improve energy and muscle mass during resistance-training.
    The purpose of this examine was to determine whether these dietary supplements affect coaching diversifications
    and/or markers of muscle anabolism/catabolism in resistance-trained athletes.
    It is possible that the problem of enhancing muscular function could be bypassed by integrating
    MSTN inhibition therapy with exercise. Research in mice show
    that combining MSTN inhibition remedy with exercise, be it cardio or resistance coaching, ends in considerably enhanced muscle high quality
    in comparability with both intervention alone [111, 112].
    Nonetheless, trials involving the mix of bimagrumab with an exercise
    program in sarcopenia sufferers, as beforehand talked about, didn’t yield any discernible difference between groups receiving mixed therapy or exercise alone [54].
    Further scientific analysis is needed to determine if the combination of MSTN inhibition therapy
    with exercise could possibly be effective in growing muscle operate in humans.

    Alternatively, follistatin 344 can be utilized with one of the SARMs referred to as ostarine.

    Myostatin is acknowledged as a significant inhibitor of muscle growth
    and its SkM and/or blood ranges are reported
    to extend with aging and in muscle losing ailments [8].
    An different means of inhibiting myostatin effects is to increase follistatin, which
    inhibits myostatin exercise by attaching to it and interfering with receptor binding [8].

    A molecular signature according to stimulatory results on SkM could
    additionally be represented by the upregulation of
    protein ranges of modulators of differentiation corresponding
    to MEF2A, Myf5, MyoD and myogenin [10]. In an try to look at the potential
    of Epi to exert optimistic results, we in contrast the
    relative ranges of the above-referred endpoints in younger vs.
    older cohorts of mice and their responses to remedy.
    Older mice demonstrated a collection of significant changes in all endpoints suggesting favorable shifts for modulators of SkM
    progress, differentiation and markers of aging (SA-β-Gal) [9].

    These findings indicate that Ac-MIF1 and Ac-MIF2-NH2 may be valuable therapeutic
    agents for the therapy of muscle-related diseases. Analysis, together with
    randomized clinical trials with each scientific and sports activities populations, in addition to these with muscle-wasting situations like sarcopenia,
    provides combined outcomes on myostatin inhibitors.
    These research examine how food regimen dietary
    supplements, nutraceuticals, and exercise affect muscle tissues
    and blood myostatin. The findings present varied results—some show decreases in myostatin levels, others
    present will increase, and some find no effect [1].
    Dark chocolate extracts and similar flavonol-containing extracts have been studied because of their talents to increase train efficiency.
    The particular flavanol, (–)-epicatechin, has been identified to extend
    nitric oxide production, enhance mitochondrial biogenesis, improve
    angiogenesis, decrease myostatin, enhance follistatin, and increase exercise performance in rodents.

    Focusing On myostatin emerged as an attractive strategy to forestall bone loss and fractures in addition to promoting muscle
    progress. Certainly, evidence from each animal and human research suggests that myostatin exerts a crucial
    impression on bone metabolism and its inhibition could additionally be useful for
    bone turnover [8,22,23]. The potent anabolic impact of
    myostatin inhibition on musculoskeletal tissues is supported by knowledge
    exhibiting that ACVR2B-Fc injection enhances both bone and
    muscle mass in wild-type and OI mouse fashions [38]. Likewise,
    systemic supply of ACVR2A-Fc elevated bone formation, bone mass, and bone strength in both regular and ovariectomized mice [54].
    It is expressed by animal and human skeletal muscle cells where it
    limits muscle growth and promotes protein breakdown.
    Computer-aided drug design is broadly regarded in the context of
    drug growth [23], and the discovery of promising lead compounds
    using these approaches offers an efficient technique
    of designing compounds with the required therapeutic profiles [24].
    Peptides are short-chain amino acids with prodigious characteristics,
    similar to excessive specificity and membrane penetration effectivity,
    and as properly as, they are fairly inexpensive [25,26].
    For these purposes, the design of peptides that mimic specific proteins has enormous therapeutic potential.
    Peptides have already had main impacts on the pharmaceutical industry and the directions of biological and chemical analysis [27].

    This change can additionally be correlated to the ability of follistatin 344 to inhibit myostatin. The over-activity of myostatin can lead
    to a situation referred to as skeletal muscle fibrosis (5).
    Follistatin is a glycoprotein produced by folliculostellate cells (FS) located within the anterior
    pituitary gland and follows the autocrine signaling pathway.

    This research confirmed a light and variable improvement in mobility in geriatric and senior canine
    with the administration of Fortetropin as decided by the LOAD mobility scores.
    Further studies will want to reveal the clinical relevance of the findings of this
    research and which canines will benefit most from Fortetropin supplementation. There
    was no important distinction in age, weight, or LOAD mobility scores at
    Weeks 0, 6, or 12 between the 2 groups (Table 2).

    Omega-3 has been linked to the biogenesis of mitochondria, selling NRF1 and NRF2, a end result ascribed to enhanced
    transcription and deacetylation of PGC-1, initiated by SIRT1 [187].
    Animals with cancer cachexia treated with omega-3
    exhibited an elevation in MAFbx and MuRF1 ranges, alongside
    a discount in the pro-inflammatory cytokine TNF-α [188].
    However, research regarding the affect of a quantity
    of dietary elements on the metabolism of muscle tissue and the modulation of MAFbx and MuRF1 by way
    of the inhibition of NRF2 stimulation by NF-κB (mostly examined in preclinical setting) has proven incongruous findings.
    MSTN is expressed in adipose and muscle tissues and performs a significant function throughout adipogenesis, which it could possibly inhibit or enhance, depending on the
    scenario [21]. Reportedly, MSTN regulates the adipogenesis of mesenchymal stem cells during the differentiation and dedication phases [22], and in animals, its
    deletion or inhibition enhances muscle mass and reduces
    fats mass [21]. One Other novel discovering of this examine was that UA remedy decreased MSTN mRNA expression by ~ 14%.

    Cocoa extracts that include epicatechin may help in promoting higher blood move and in enhancing the cells sensitivity to insulin. In vitro and in vivo experiments were accomplished on Leydig cells (cells responsible for producing testosterone).
    IGF-1 LR3 works by making muscles more sensitive to insulin which leads to the building of muscle tissues.

    What separates follistatin 344 from the the rest of the proteins is
    that relatively less consumption can convey long-lasting effects on the body.
    Although follistatin 344 is a recombinant spinoff of naturally occurring follistatin,
    the dearth of human-based clinical trials still makes it an illegal substance
    for people to eat.
    It utilizes an engineered scaffold primarily based on the tenth fibronectin sort III domain coupled with a human IgG Fc
    domain, exhibiting a binding affinity for the C-terminal of mature MSTN
    and the ActRIIB–MSTN complicated, just like
    an antibody [69]. Taldefgrobep binding prevents ALK-4/5
    recruitment, thereby inhibiting the SMAD pathway.
    In a part 1 trial involving healthy adults, a 24-week therapy interval with weekly doses starting from forty five to 150 
    mg led to a universal lower in free MSTN by ≥ 90%.
    Additionally, important increases in lean physique mass,
    up to a mean enhance of 2.69%, and thigh muscle volume,
    as much as a mean improve of four.75%, have been observed [69].

    In a mouse mannequin for rheumatoid arthritis, MSTN is highly expressed in synovial tissues,
    and transgenic or antibody inhibition of MSTN ameliorates joint destruction and arthritis severity [149].
    Although MSTN inhibition has proven promise in animal fashions for
    increasing bone mass and improving bone strength, no
    clinical trials concentrating on orthopedic
    ailments have been performed. The just lately
    described enhance in axon number together with delay in age-related neural degeneration in myostatin null mice have added assist to the investigation of myostatin inhibitors for the treatment of extreme neuromuscular
    disorders [54▪,55].
    Administration of this peptide to MDX mice (a mannequin of DMD)
    significantly increased muscle mass. Sulfo-polysaccharide is a
    nutrient that’s advertised to bind to myostatin and inhibit its exercise in muscle.

    Sulfo-polysaccharide’s energetic ingredient is a brown sea algae known as
    cystoseira canariensis.
    Similarly, GYM-329 by Roche is designed to deal with FSHD by binding
    to latent MSTN, thus blocking its conversion to its mature kind [61].
    These antibodies are presently present process medical trials or awaiting the reporting of
    results. The majority of clinically tested MSTN inhibitors have been MSTN-based antibodies.

    Ultimately, the creator emphasised that only a few studies endorsed the impact of non-protein supplements on myostatin,
    and their use stays unsupported. The full cycle has been proven in studies by injecting mice
    with myostatin, seeing them enter advanced muscle wastage, and then reversing it by administering one of many myostatin inhibitors (e.g.
    follistatin or myostatin-propeptide). Glad you requested, because that’s one
    of the issues with oversimplifying this problem. Knocking out the myostatin gene at start
    or inhibiting the protein it encodes later in life seems to
    positively affect muscle development, in mice a minimum
    of. Aside from the truth that the athletic neighborhood views myostatin inhibition as cheating, the scientific
    analysis about its adverse results for athletic enhancement is sparse.

    We discovered that ingestion of FOR prevented the rise in circulating plasma
    myostatin. Nevertheless, FOR supplementation did not attenuate the lack of muscle CSA measured by
    ultrasonography, muscle fiber CSA in kind I and II, and
    LM of the immobilized leg. In addition, both groups didn’t
    get well the immobilization-induced reduction in muscle CSA and LM of the immobilized leg after two weeks of returning
    to normal physical activity. Isometric peak torque decreased
    following two weeks of single-leg immobilization;
    however, two weeks of returning to normal exercise was enough to recover the discount in peak torque.
    Forty-five resistance-trained males who had been constantly coaching for
    no much less than one yr volunteered for this study.
    Topics had been considered resistance educated if they had been consistently
    training for one yr, a minimum of three days per week.
    This not solely aids in muscle recovery and progress but additionally enhances the effectiveness of different dietary supplements by bettering their bioavailability.
    This regulatory mechanism ensures that muscle development doesn’t go unchecked, which is crucial for normal growth.
    MightyBecause they wished to know for positive whether or not the Mighty
    gene is essential to muscle progress the researchers did
    one other experiment. The figures under show that
    within the muscle cells where this occurred [shRNA Akirin-1] fewer muscle fibres were indeed shaped than in muscle cells by which the
    Mighty gene was nonetheless lively [shRNA scrambled].
    Exercise-induced mitochondrial biogenesis happens when skeletal muscle cells create
    new mitochondria in response to strenuous exercise.
    As a results of muscle mitochondrial content growing, basal metabolic fee rises, oxidative capability expands, and various
    other advantageous variations take place [21].
    Past the primary regulatory choice for apitegromab, corporations are pivoting
    to the larger potential medical and commercial alternative for muscle mass preservation in overweight and
    overweight people taking the wildly well-liked incretin mimetic weight-loss medication. There
    are a selection of intervention factors being exploited to inhibit myostatin signaling in order to improve muscle mass underneath the circumstances of muscle atrophy.
    Results of a number of Part 2 trials underway are awaited to determine if elevated muscle volumes translate into muscle energy, efficiency
    and outcomes which may be clinically meaningful to sufferers.
    Perhaps not surprisingly skeletal muscle tumors, particularly rhabdomyosarcomas (RMS),
    the commonest delicate tissue tumor in children, are known to overexpress myostatin [92].
    Blocking myostatin activity with a dominant adverse type of ActRIIB resulted in decreased proliferation and promoted differentiation of a human RMS cell line suggesting that myostatin inhibition could additionally be a priceless target for interventions for RMS [93].

    More apparently, Lokireddy et al.[94▪▪] for the primary time reported that myostatin protein is expressed and secreted from the mouse adenocarcinoma cell line C26, and from several
    human cancer cell traces.
    The number of MHC-IIx and MHC-IIb expressing
    fibers in the soleus was noticeably elevated with HS (Fig.
    5D); however, fluorescent staining was inconsistent and generally weak as compared to the strongly stained
    IIx and IIb fibers in the gastrocnemius. As a result, it proved somewhat difficult to quantify
    CSA or fiber kind distribution especially in soleus IIb fibers.

    As you embark in your fitness journey, think about the dear function that these
    dietary supplements can play in unlocking your muscle-building potential.
    Embrace the science-backed advantages of myostatin inhibition, and take assured steps in path
    of attaining your health objectives. Bear In Mind, supplements
    are highly effective tools in your arsenal, providing you the
    support you should sculpt the physique you need.
    Additionally, EP1LOGUE is certainly one of the few obtainable
    Urolithin B-focused dietary supplements.

    On the opposite hand, serum MSTN ranges are elevated in aged people and inversely correlated with lean mass.
    This discordance could stem from the technical limitations
    of enzyme-linked immunosorbent assays or radioimmunoassays used to discriminate between active and inactive
    MSTN or between MSTN and the similar protein (GDF-11) (Yarasheski et al., 2002).
    In SM, although MSTN mRNA levels usually are not correlated with
    age, MSTN protein levels are elevated in aged topics.
    Furthermore, MSTN protein ranges are higher in the muscle tissues of elderly males than in wholesome younger males after acute muscle train (McKay et al., 2012).
    Sarcopenia is the result of a decline in the number of motor items and
    muscle fiber atrophy and is more prevalent today as a end result of rising number of elderly (Stoever et al., 2017).

    Although it’s well known that reduced protein synthesis and/or elevated protein degradation induces SM
    atrophy, stories concerning the underlying molecular pathways are
    inconsistent. However, numerous stories have demonstrated that MSTN is a possible therapeutic goal (White and LeBrasseur, 2014).

    Leptin signaling, through the hypothalamic melanocortin receptors,
    acts as a possible mediator of inflammation-induced cachexia and anorexia in CKD.
    Kidneys clear the circulating leptin, therefore, its serum concentration is elevated in uremia and CKD [143].
    Leptin is taken into the mind by the blood-brain barrier (BBB) and binds to the leptin receptor,
    suppressing meals intake by inhibiting the arcuate nucleus of
    the hypothalamus. Leptin controls cachexia by way of modifying vitality expenditure and meals intake by way of the hypothalamic melanocortin system [21].
    In CKD sufferers, the decreased renal function leads
    to an elevation in leptin levels, in all probability due to diminished renal clearance, thus, increased levels of leptin are related to poor dietary statuses, corresponding to hypercatabolism and low serum albumin [9, 144].
    In the hypothalamic arcuate nucleus neurons,
    Proopiomelanocortin (POMC, a pro-peptide precursor) is produced that impedes power storage and food consumption by releasing α–melanocyte-stimulating hormone (α-MSH)
    [145]. Α-MSH prohibits food consumption by binding to melanocortin receptors-3 and − four (MC3-R
    and MC4-R), mainly through MC4-R [134].

    References:

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  61. how long does it take for steroids to Get Out of your system says:

    It might or may not be right for you, and you’ll discover out extra about that within the guide
    under. By exploring these sections on Anavar stacking options, people can acquire insights into
    how combining Anavar with other compounds can improve their desired outcomes.
    Moreover, it’s essential to prioritize proper nutrition, common exercise,
    and adequate relaxation through the cycle to optimize results and reduce potential risks.
    Combining Anavar with a balanced food regimen and an appropriate
    coaching program can yield optimistic effects on muscle
    definition, power, and overall physique. In this part, we are going to present a
    comprehensive overview of an Anavar instance cycle, which serves as a roadmap for individuals
    contemplating using this in style anabolic steroid. Anavar cycles are carefully deliberate periods of
    time during which the steroid is taken, with specific dosages
    and durations in mind. Anavar’s half life is 8 hours, which is relatively brief in comparison to different steroids.

    This is a friendly contest prep stack suited for hardcore
    users and competitors. Winstrol poses the next liver toxicity and aspect effect risk
    than Anavar, so we will hold this stack brief. Doses are around
    half that of what we’d normally use for every compound because they’re both oral steroids.
    All of those stacks would require a post-cycle remedy protocol afterward.
    A normal PCT is Clomid at 100mg daily, starting two weeks after
    your final dose. While Clenbuterol suppresses the appetite, Anavar can enhance hunger
    in some users. Anavar at 10mg/day for 6-8 weeks is sweet, and also you in all
    probability won’t have any unwanted facet effects.

    Clenbuterol can additionally be stacked with Cytomel (synthetic T3) to reinforce fats burning.
    Cytomel is a prescription medicine for hypothyroidism (an underactive thyroid).
    Insomnia could be skilled by clenbuterol users more
    vulnerable to an overly lively nervous system, inflicting hyperactivity
    (4). Tremors are also common when taking clenbuterol due to its greater adrenaline output.

    As with any medication or supplementation, it is essential to approach Anavar with an knowledgeable perspective.
    For example, if you are experiencing abdomen upset, you might wish to take Anavar with food or at a different time of day.

    If you may be experiencing insomnia, you might want to keep away from taking Anavar earlier than mattress.
    For Anavar, this means it usually has a brief half-life around 8-14 hours.
    This interprets in layman’s phrases to “how long does something stay in your system”; regardless of age or different factors, Anavar won’t stick around longer than 14 hours from
    utility.
    Nevertheless, it’s important to exercise caution when utilizing liquid Anavar to ensure correct dosing, as measuring liquid volumes can be less
    exact than merely taking a tablet or pill. Tablets and tablets are
    the most prevalent and user-friendly forms of Anavar. Anavar pills and tablets are sometimes obtainable in standardized doses, allowing customers to have better management over their consumption. This kind is especially
    appealing to people who prioritize convenience and ease in their complement
    regimen. The well-defined dosage additionally makes
    it appropriate for each newbies and experienced customers,
    because it eliminates the need for measuring or calculating doses.

    While oral administration provides comfort and ease of
    use, it also poses some disadvantages. The
    potential for liver toxicity is a major concern, as the liver must process the compound, which may exert
    pressure on this important organ. Additionally, oral
    Anavar could have a shorter half-life compared to different administration methods, requiring extra frequent dosing.
    Anavar is primarily eliminated by way of renal excretion following hepatic metabolism.
    After oral administration, it undergoes hydroxylation and
    conjugation within the liver, increasing water solubility for kidney excretion.
    Nonetheless, those that don’t get hit hard by crazy
    pumps can make some pace features with var because it will increase muscle efficiency without including weight.
    If you’re missing motivation because of lessened strength and muscle
    positive aspects, then you possibly can actually kickstart things in the gym with var.
    Analysis suggests that many drugs can remain potent
    for a minimal of five years after the expiration date.
    This is before utilizing expired medication to ensure security
    and effectiveness. Sure, just like all anabolic steroids and medicines,
    Anavar does go dangerous. It represents the maker’s assurance that the treatment stays
    secure and potent until that date.
    Sorry to bust out the cliché get-ripped-by-summer topic that’s so common in the
    health trade. Hello, I Am Dave Moffat the founder and Chief Editor of steroidsourcetalk.com and certified Worldwide Personal Trainer and Licensed
    Nutritionist. My passion has all the time been bodybuilding but with 15 years’ expertise
    in weight reduction packages too, it’s hard to
    not mention all that if you’re working at your health level fullest (I hope).

    When Im not in the gym or spending time away from my household i typically think about
    what advice would help others achieve theirs objectives identical to these
    impressed mine. Anavar is well known as the one anabolic steroid girls can use with little to no threat of side effects
    – as lengthy as doses don’t exceed 20mg per day.
    However, some other compounds are additionally utilized by females,
    together with Winstrol and Primobolan, as nicely as the fat-burning compound Clenbuterol.
    Most ladies will use Anavar by itself in a cycle, however additionally it is generally stacked
    with a fat-burning compound known as Clenbuterol (not an anabolic
    steroid).
    Anavar can affect hormone ranges, together with natural testosterone manufacturing.
    Both oral and sublingual administration strategies can suppress endogenous testosterone production, leading to hormonal imbalances.
    Post-cycle therapy (PCT) may be necessary to restore normal
    testosterone ranges after discontinuing Anavar. When it comes to enhancing energy and endurance,
    Anavar can be a game-changer. Oral and sublingual administration methods can both contribute to elevated energy output
    and stamina. By enhancing red blood cell manufacturing and enhancing oxygen-carrying capability, Anavar
    helps athletes push by way of intense workouts and attain new performance heights.

    Anavar can produce some mild androgenic results in men, and hair loss can be one of these points for
    males who’ve a genetic predisposition to baldness. Outstanding fats loss might be seen on this stack, and it’ll come on quickly.

    Count On a rise in energy and endurance, however the unwanted aspect effects of Clen can hurt your train capacity (lowering the dose is ideal if you’re sensitive to stimulants).
    Anavar will present the capacity to construct muscle and maintain strength while dieting.
    A beginner can safely run an 8-week cycle (the most beneficial length) at a dosage of 30-50mg daily.
    A typical PCT protocol after an Anavar cycle could contain the use of selective estrogen receptor modulators (SERMs) corresponding to tamoxifen or clomiphene.
    The duration and dosage of the PCT will range
    based on the size and dosage of the Anavar cycle, in addition to individual factors.

    Carrying a really mild androgenic nature with nearly no androgenic activity.
    Anavar, often referred to as “Var” or “A-var” when discussing it’s use
    as a sports efficiency enhancer. This article covers
    the legal anavar complement, as nicely as the previous a-var
    which contained oxandrolone. If you are stacking Anavar with different steroids, you will need to begin with a lower dosage of Anavar and increase it progressively.
    It can take a couple of weeks for the drug to build up in your system and produce results.
    However, some folks do expertise advantages within the first week or two of taking it.

    References:

    how long does it take for steroids to Get Out of your system

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